Myo1f is critical for neutrophil migration in vivo

Página do item simplificado
dc.creatorMauro Martins Teixeira
dc.date.accessioned2021-08-02T10:08:26Z
dc.date.accessioned2025-09-08T23:29:29Z
dc.date.available2021-08-02T10:08:26Z
dc.date.issued2018-04-26
dc.format.mimetypepdf
dc.identifier.doihttps://doi.org/10.1182/blood-2018-03-837872
dc.identifier.issn15280020
dc.identifier.urihttps://hdl.handle.net/1843/37157
dc.languageeng
dc.publisherUniversidade Federal de Minas Gerais
dc.relation.ispartofBlood
dc.rightsAcesso Aberto
dc.subjectNeutrófilos
dc.subjectInfecções
dc.subjectMiosinas
dc.subject.otherNeutrophils
dc.subject.otherPolymorphonuclear leukocytes
dc.subject.otherInfection
dc.subject.otherMyo1f
dc.titleMyo1f is critical for neutrophil migration in vivo
dc.typeArtigo de periódico
local.citation.epage1880
local.citation.issue17
local.citation.spage1879
local.citation.volume131
local.description.resumoIn this issue of Blood, Salvermoser et al demonstrate that the unconventional class I myosin 1f (Myo1f) is crucial for neutrophil trafficking during acute inflammation in vascular beds in vivo. Importantly, they demonstrate that Myo1f is crucial for the dynamics of the deformation of the neutrophil nucleus and consequent capacity of these cells to extravasate in vivo and to negotiate through three-dimensional (3D) environments.
local.identifier.orcidhttps://orcid.org/0000-0002-6944-3008
local.publisher.countryBrasil
local.publisher.departmentICB - DEPARTAMENTO DE BIOQUÍMICA E IMUNOLOGIA
local.publisher.departmentICB - DEPARTAMENTO DE FARMACOLOGIA
local.publisher.initialsUFMG
local.url.externadoi:10.1182/blood-2018-03-837872

Arquivos

Pacote original

Agora exibindo 1 - 1 de 1
Imagem de Miniatura
Nome:
Myo1f is critical for neutrophil migration in vivo.pdf
Tamanho:
724.93 KB
Formato:
Adobe Portable Document Format
Baixar

Licença do pacote

Agora exibindo 1 - 1 de 1
Imagem de Miniatura
Nome:
License.txt
Tamanho:
1.99 KB
Formato:
Plain Text
Descrição:
Baixar

Coleções

Artigo de Periódico

Logo UFMG