Atorvastatin reduced soluble receptors of TNF-alpha in systemic lupus erythematosus

dc.creatorGilda Aparecida Ferreira
dc.creatorA. L. Teixeira
dc.creatorDébora Cerqueira Calderaro
dc.creatorEmília Inoue Sato
dc.date.accessioned2023-07-19T20:27:38Z
dc.date.accessioned2025-09-08T23:01:53Z
dc.date.available2023-07-19T20:27:38Z
dc.date.issued2016
dc.format.mimetypepdf
dc.identifier.issn1593098X
dc.identifier.urihttps://hdl.handle.net/1843/56768
dc.languageeng
dc.publisherUniversidade Federal de Minas Gerais
dc.relation.ispartofClinical and Experimental Rheumatology
dc.rightsAcesso Aberto
dc.subjectLúpus Eritematoso Sistêmico
dc.subjectAterosclerose
dc.subjectAtorvastatina
dc.subject.otheraterosclerose
dc.subject.otheratorvastatin
dc.subject.othersystemic lupus erythematosus
dc.titleAtorvastatin reduced soluble receptors of TNF-alpha in systemic lupus erythematosus
dc.typeArtigo de periódico
local.citation.epage48
local.citation.issue1
local.citation.spage42
local.citation.volume34
local.description.resumoThe aim of this study was to evaluate the efficacy of atorvastatin to reduce the plasma levels of TNF system molecules(TNF-α, sTNFR1 and sTNFR2) and to assess their association with risk factors for accelerate atherosclerosis and clinical disease activity scores in SLE patients.In a previous study, 64 female SLE patients received 20 mg/day of atorvastatin and 24 SLE patients (non-treated group)were followed for 8 weeks. Plasma levels of TNF-α, sTNFR 1 and sTNFR 2 were measured by ELISA, at baseline and atthe end of the study.The plasma levels of sTNFR1 and sTNFR 2 showed a positive correlation with SLEDAI score. We also found a positivecorrelation between TNF-α and sTNFR 1 levels and SLICC score. Patients with current nephritis and patients with anti-dsDNA antibodies presented higher sTNFR1 and sTNFR2 levels. Patients with abdominal obesity and arterial hypertension also had higher plasma levels of soluble receptors. At the end of 8 weeks, we observed a significant decrease in sTNFR1 plasma levels in patients receiving atorvastatin [median (percentile), 876.5 (717–1284 pg/ml) vs. 748 (629.6–917.3 pg/ml),p=0.03], without difference regarding TNF-α and sTNFR2 plasma levels. The SLEDAI and SLICC scores were independent determinants of the plasma levels of sRTNF1.Atorvastatin reduced soluble receptors of TNF-α. The plasma levels of TNF-α, sTNFR1 and sTNFR2 may play a role in SLE activity and atherosclerosis, and might be evaluated as targets for new therapies.
local.publisher.countryBrasil
local.publisher.departmentMED - DEPARTAMENTO DE APARELHO LOCOMOTOR
local.publisher.departmentMED - DEPARTAMENTO DE CLÍNICA MÉDICA
local.publisher.initialsUFMG
local.url.externahttps://www.clinexprheumatol.org/abstract.asp?a=9060

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