Surgical removal of giantform unilateral mandibular tori: a case report

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Universidade Federal de Minas Gerais

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Artigo de periódico

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Background: Mandibular and palatine tori it's an exostosis formed by a dense cortical and a reduced amount of medular bone covered by a tiny and poorly vascularized mucosa. Commonly diagnosed during middle age, not presenting large proportions, fast growing or malignant evolution. Recommended it's surgical removal when frequently injured or presenting occlusal interference. The justification for it's maintenance it's the fact that it could be used as an autogenous graft in periodontics and implantology. Objectives: The present work aims to report a surgical removal of a giantform unilateral mandibular tori. Methods: Patient R. A. G., male, 34 years old, feoderma, appeared on our service complaining of pain, frequent ulcerations, speech problems and teeth movement. Report that have noticed the lesion's appearance at the age of thirteen and the same has been growing slowly since then. On clinical examination, it was noticed extensive and unilateral tumor lesion on the lingual right side of the mandible's body and on radiographic examination, multilobulated radiopaque lesion similar to the adjacent cortical bone projecting to the median line. It's was opted for surgical removal under local anesthesia, realized mucoperiosteal detachment on the lingual side and it's exeresis was realized by osteotomies with segmentation and wear drills. Findings: The amount of bone removed was sufficient to end the patient's complaints and was limited by the portion above the miloid line. There were no complications. The bone fragments’ histopathological examination presented the diagnosis of lamellar bone tissue. Conclusion: It's important to perform a complete oral evaluation when removing such lesion, since the same can be used as an autogenous bone graft

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Exostoses, General surgery, Neoplasms, Mandible, Radiography, Osteotomy

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giantform unilateral mandibular

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https://www.ijoms.com/article/S0901-5027(19)30797-0/fulltext

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