Immunology and immunopathogenesis of human leishmaniasis

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dc.creatorJuliana P. B. de Menezes
dc.creatorCláudia Brodskyn
dc.creatorRicardo Gonçalves
dc.creatorOlivia Bacellar
dc.date.accessioned2023-11-22T22:07:36Z
dc.date.accessioned2025-09-09T01:20:46Z
dc.date.available2023-11-22T22:07:36Z
dc.date.issued2022
dc.format.mimetypepdf
dc.identifier.doihttps://doi.org/10.3389/fcimb.2022.1055221
dc.identifier.issn2235-2988
dc.identifier.urihttps://hdl.handle.net/1843/61292
dc.languageeng
dc.publisherUniversidade Federal de Minas Gerais
dc.relation.ispartofFrontiers in Cellular and Infection Microbiology
dc.rightsAcesso Aberto
dc.subjectLeishmania
dc.subjectPioglitazona
dc.subjectPatogênese Homeopática
dc.subject.otherHuman leishmaniasis
dc.subject.otherImmunopathogenesis
dc.subject.otherIL-32
dc.subject.otherIL-17
dc.subject.otherPeroxisome proliferator-activated receptor-g (PPAR-g),
dc.subject.otherPioglitazone
dc.titleImmunology and immunopathogenesis of human leishmaniasis
dc.typeArtigo de periódico
local.citation.epage4
local.citation.spage1055221
local.citation.volume12
local.description.resumoLeishmaniasis is a disease that affects people and animals worldwide and can be broadly divided into visceral (VL) and tegumentary (TL) forms. TL caused by Leishmania braziliensis presents a wide spectrum de clinical manifestations ranging from a single and typical ulcer known as CL to the involvement of nasal or oral mucosal, mucosal leishmaniasis (ML). Between both clinical forms, there is diffuse cutaneous leishmaniasis (DCL) caused by Leishmania amazonensis. This spectrum included atypical cutaneous leishmaniasis (ACL) characterized by vegetative, verrucous, crusted and lupoid lesions (Guimarães et al., 2016). Human VL is characterized by depressed cell-mediated immunity and decreased Th1 immune response (Carvalho et al., 1989; Bacellar et al., 2000). Also, VL is associated with increased production of multiple pro-inflammatory cytokines and chemokines (Saporito et al., 2013; Palacios et al., 2021; Tasew et al., 2021).
local.identifier.orcidhttps://orcid.org/0000-0002-1127-4483
local.publisher.countryBrasil
local.publisher.departmentICB - DEPARTAMENTO DE PATOLOGIA
local.publisher.departmentICB - INSTITUTO DE CIÊNCIAS BIOLOGICAS
local.publisher.initialsUFMG
local.url.externahttps://www.frontiersin.org/articles/10.3389/fcimb.2022.1055221/full

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